换一批
换一批Juvenile Huntington's Disease Skin Fibroblasts Respond with Elevated Parkin Level and Increased Proteasome Activity as a Potential Mechanism to Counterbalance the Pathological Consequences of Mutant Huntingtin Protein.
作者：
主题词
细胞增殖(Cell Proliferation);细胞, 培养的(Cells, Cultured);成纤维细胞(Fibroblasts);GTP磷酸水解酶类(GTP Phosphohydrolases);糖酵解(Glycolysis);人类(Humans);杭廷顿病(Huntington Disease);膜电位, 线粒体(Membrane Potential, Mitochondrial);线粒体(Mitochondria);线粒体膜转运蛋白质类(Mitochondrial Membrane Transport Proteins);突变(Mutation);神经元(Neurons);肽类(Peptides);蛋白酶体内肽酶复合物(Proteasome Endopeptidase Complex);活性氧(Reactive Oxygen Species);皮肤(Skin);泛素(Ubiquitin);泛素蛋白连接酶类(Ubiquitin-Protein Ligases)
DOI
10.3390/ijms20215338
PMID
31717806
发布时间
2020-04-13
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