Siglec-G Deficiency Ameliorates Hyper-Inflammation and Immune Collapse in Sepsis via Regulating Src Activation.
第一作者:
Wenqian,Li
第一单位:
Department of Anesthesiology, Changzheng Hospital, Second Military Medical University, Shanghai, China.
作者:
主题词
动物(Animals);细胞因子类(Cytokines);酶激活(Enzyme Activation);缺氧诱导因子1, α亚基(Hypoxia-Inducible Factor 1, alpha Subunit);炎症(Inflammation);白细胞介素10(Interleukin-10);外源凝集素类(Lectins);巨噬细胞(Macrophages);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);NF-κB(NF-kappa B);受体, 抗原, B细胞(Receptors, Antigen, B-Cell);含SH2域蛋白质酪氨酸磷酸酶类(SH2 Domain-Containing Protein Tyrosine Phosphatases);STAT3转录因子(STAT3 Transcription Factor);脓毒症(Sepsis);信号传导(Signal Transduction);Toll样受体(Toll-Like Receptors);src族激酶类(src-Family Kinases)
DOI
10.3389/fimmu.2019.02575
PMID
31781099
发布时间
2020-12-10
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