Sp1-independent downregulation of NHEJ in response to BER deficiency.

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第一作者： Polina S,Loshchenova

第一单位： Institute of Cytology and Genetics, Russian Academy of Sciences, Lavrentieva 10, Novosibirsk, 630090, Russian Federation; Novosibirsk State University, Pirogova 2, Novosibirsk, 630090, Russian Federation.

作者： Polina S,Loshchenova ^[1] ; Svetlana V,Sergeeva ^[1] ; Dmitry V,Limonov ^[1] ; Zhigang,Guo ^[2] ; Grigory L,Dianov ^[3]

作者单位： Institute of Cytology and Genetics, Russian Academy of Sciences, Lavrentieva 10, Novosibirsk, 630090, Russian Federation; Novosibirsk State University, Pirogova 2, Novosibirsk, 630090, Russian Federation. ^[1] Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 WenYuan Road, Nanjing, 210023, China. ^[2] Institute of Cytology and Genetics, Russian Academy of Sciences, Lavrentieva 10, Novosibirsk, 630090, Russian Federation; Novosibirsk State University, Pirogova 2, Novosibirsk, 630090, Russian Federation; Jiangsu Key Laboratory for Molecular and Medical Biotechnology, College of Life Sciences, Nanjing Normal University, 1 WenYuan Road, Nanjing, 210023, China; Oxford Institute for Radiation Oncology, Department of Oncology, University of Oxford, Old Road Campus Research Building, Oxford, OX3 7DQ, United Kingdom. Electronic address: grigory.dianov@oncology.ox.ac.uk. ^[3]

关键词 Base excision repair (BER)DNA damage Genome stability Non-homologous End joining (NHEJ)Transcription factor Sp1