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Noncanonical autophagy in dermal dendritic cells mediates immunosuppressive effects of UV exposure.

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第一作者： Payel,Sil

第一单位： Immunity, Inflammation, and Disease Laboratory, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, NC.

作者： Payel,Sil ^[1] ; Jutamas,Suwanpradid ^[2] ; Ginger,Muse ^[1] ; Artiom,Gruzdev ^[3] ; Liwen,Liu ^[4] ; David L,Corcoran ^[5] ; Cynthia J,Willson ^[6] ; Kyathanahalli,Janardhan ^[6] ; Sara,Grimm ^[7] ; Page,Myers ^[8] ; Laura Miller,Degraff ^[1] ; Amanda S,MacLeod ^[9] ; Jennifer,Martinez ^[10]

作者单位： Immunity, Inflammation, and Disease Laboratory, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, NC. ^[1] Department of Dermatology, Duke University, Durham, NC. ^[2] Knockout Mouse Core Laboratory, Reproductive and Developmental Biology Laboratory, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, NC. ^[3] Molecular Genomics Core Laboratory, Signal Transduction Laboratory, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, NC. ^[4] Duke Center for Genomic and Computational Biology, Duke University Medical Center, Durham, NC. ^[5] Integrated Laboratory Systems, Inc, Research Triangle Park, NC. ^[6] Division of Intramural Research, Research Triangle Park, NC. ^[7] Comparative Medicine Branch, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, NC. ^[8] Department of Dermatology, Duke University, Durham, NC; Department of Immunology, Duke University, Durham, NC; Department of Molecular Genetics and Microbiology, Duke University, Durham, NC. ^[9] Immunity, Inflammation, and Disease Laboratory, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, NC. Electronic address: jennifer.martinez3@nih.gov. ^[10]

关键词 Autophagy LAP Rubicon antigen presentation contact hypersensitivity dendritic cells efferocytosis inflammation