aYAP modRNA reduces cardiac inflammation and hypertrophy in a murine ischemia-reperfusion model.-论文-万方医学网

第一作者： Jinmiao,Chen

第一单位： Boston Children's Hospital, Boston, MA, USA.

作者： Jinmiao,Chen ^[1] ; Qing,Ma ^[2] ; Justin S,King ^[1] ; Yan,Sun ^[1] ; Bing,Xu ^[3] ; Xiaoyu,Zhang ^[3] ; Sylvia,Zohrabian ^[1] ; Haipeng,Guo ^[1] ; Wenqing,Cai ^[1] ; Gavin,Li ^[4] ; Ivone,Bruno ^[5] ; John P,Cooke ^[1] ; Chunsheng,Wang ^[6] ; Maria,Kontaridis ^[6] ; Da-Zhi,Wang ^[2] ; Hongbo,Luo ^[3] ; William T,Pu ^[1] ; Zhiqiang,Lin ^[1]

作者单位： Boston Children's Hospital, Boston, MA, USA. ^[1] Department of Cardiovascular Surgery and Shanghai Institute of Cardiovascular Disease, Zhongshan Hospital, Fudan University, Shanghai, China. ^[2] Masonic Medical Research Institute, Utica, NY, USA. ^[3] Department of Critical Care Medicine, Key Laboratory of Cardiovascular Remodeling and Function Research, Chinese Ministry of Education and Chinese Ministry of Health, Qilu Hospital of Shandong University, Jinan, China. ^[4] Boston Children's Hospital and Dana Farber Cancer Institute, Boston, MA, USA. ^[5] Houston Methodist Research Institute, Houston, TX, USA. ^[6] Boston Children's Hospital, Boston, MA, USA wpu@pulab.org. ^[7] Harvard Stem Cell Institute, Cambridge, MA, USA. ^[8] Boston Children's Hospital, Boston, MA, USA zlin@mmri.edu. ^[9]

主题词衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);动物(Animals);动物, 新生(Animals, Newborn);细胞凋亡(Apoptosis);心脏扩大(Cardiomegaly);细胞存活(Cell Survival);细胞, 培养的(Cells, Cultured);疾病模型, 动物(Disease Models, Animal);人类(Humans);注射, 肌肉内(Injections, Intramuscular);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);心肌再灌注损伤(Myocardial Reperfusion Injury);心肌炎(Myocarditis);心肌(Myocardium);肌细胞, 心脏(Myocytes, Cardiac);中性白细胞浸润(Neutrophil Infiltration);RNA编辑(RNA Editing);RNA, 信使(RNA, Messenger);转录因子(Transcription Factors)

DOI 10.26508/lsa.201900424

PMID 31843959

发布时间 2023-10-31

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aYAP modRNA reduces cardiac inflammation and hypertrophy in a murine ischemia-reperfusion model.

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aYAP modRNA reduces cardiac inflammation and hypertrophy in a murine ischemia-reperfusion model.

Life science alliance

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