FC-99 reduces macrophage tenascin-C expression by upregulating miRNA-494 in arthritis.

导出 LinkOut

翻译打印收藏纠错

作者： Haiyan,Zhu ^[1] ; Juanhua,Fu ^[1] ; Sheng,Chen ^[2] ; Xiaoqin,Li ^[1] ; Huaping,Liang ^[3] ; Yayi,Hou ^[4] ; Huan,Dou ^[5]

作者单位： The State Key Laboratory of Pharmaceutical Biotechnology, Division of Immunology, Medical School, Nanjing University, Nanjing 210093, China. ^[1] Nangjing Stomatological Hospital, Medical School of Nanjing University, Nanjing 210008, China. ^[2] State Key Laboratory of Trauma, Burns and Combined Injury, Research Institute of Surgery, Daping Hospital, The Army Medical University, Chongqing 400042, China. Electronic address: 13638356728@163.com. ^[3] The State Key Laboratory of Pharmaceutical Biotechnology, Division of Immunology, Medical School, Nanjing University, Nanjing 210093, China; Department of Rheumatology and Immunology, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing 210008, China; Jiangsu Key Laboratory of Molecular Medicine, Nanjing University, Nanjing 210093, China. Electronic address: yayihou@nju.edu.cn. ^[4] The State Key Laboratory of Pharmaceutical Biotechnology, Division of Immunology, Medical School, Nanjing University, Nanjing 210093, China; Department of Rheumatology and Immunology, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing 210008, China; Jiangsu Key Laboratory of Molecular Medicine, Nanjing University, Nanjing 210093, China. Electronic address: douhuan@nju.edu.cn. ^[5]

关键词 FC-99 Macrophages MiRNA-494 Tenascin-C Zymosan-induced arthritis