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Inhibition deficits are modulated by age and CGG repeat length in carriers of the FMR1 premutation allele who are mothers of children with fragile X syndrome.

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第一作者： Jessica,Klusek

第一单位： Department of Communication Sciences and Disorders, University of South Carolina, 1705 College Street, Columbia, SC 29208, USA.

作者： Jessica,Klusek ^[1] ; Jinkuk,Hong ^[2] ; Audra,Sterling ^[3] ; Elizabeth,Berry-Kravis ^[4] ; Marsha R,Mailick ^[5]

作者单位： Department of Communication Sciences and Disorders, University of South Carolina, 1705 College Street, Columbia, SC 29208, USA. ^[1] Waisman Center, University of Wisconsin-Madison, 1500 Highland Ave, Madison, WI 53705, USA. ^[2] Waisman Center, University of Wisconsin-Madison, 1500 Highland Ave, Madison, WI 53705, USA; Department of Communication Sciences and Disorders, University of Wisconsin-Madison, 381 Goodnight Hall, 1975 Willow Drive, Madison, WI 53706, USA. ^[3] Department of Pediatrics, Neurological Sciences and Biochemistry, Rush University Medical Center, 1725 West Harrison Street, Suite 718, Chicago, IL 60612, USA. ^[4] Waisman Center, University of Wisconsin-Madison, 1500 Highland Ave, Madison, WI 53705, USA. Electronic address: marsha.mailick@wisc.edu. ^[5]

关键词 FMR1 premutation age effects executive dysfunction fragile X premutation carrier mid-range CGG