Hypoxia-inducible factor 1α promotes interleukin 1β and tumour necrosis factor α expression in lipopolysaccharide-stimulated human dental pulp cells.

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作者： M,Fujii ^[1] ; N,Kawashima ^[1] ; K,Tazawa ^[1] ; K,Hashimoto ^[1] ; K,Nara ^[1] ; S,Noda ^[1] ; S,Nagai ^[2] ; T,Okiji ^[1]

作者单位： Division of Oral Health Sciences, Department of Pulp Biology and Endodontics, Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University (TMDU), Tokyo, Japan. ^[1] Division of Oral Health Sciences, Department of Molecular Immunology, Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University (TMDU), Tokyo, Japan. ^[2]

关键词 HIF1α NFκB signalling human dental pulp cells lipopolysaccharide pro-inflammatory cytokine

主题词细胞, 培养的(Cells, Cultured);牙髓(Dental Pulp);人类(Humans);白细胞介素1β(Interleukin-1beta);脂多糖类(Lipopolysaccharides);NF-κB(NF-kappa B);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)

DOI 10.1111/iej.13264

PMID 31910287

发布时间 2020-04-14

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