High bone microarchitecture, strength, and resistance to bone loss in MRL/MpJ mice correlates with activation of different signaling pathways and systemic factors.

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第一作者： Xuying,Sun

第一单位： Department of Orthopaedic Surgery, The Brown Foundation Institute of Molecular Medicine, McGovern Medical School, University of Texas Health Science Center at Houston, Houston, Texas.

作者： Xuying,Sun ^[1] ; Xueqin,Gao ^[2] ; Zhenhan,Deng ^[1] ; Linlin,Zhang ^[3] ; Kirk,McGilvray ^[4] ; Benjamin C,Gadomski ^[4] ; Sarah,Amra ^[1] ; Gang,Bao ^[3] ; Johnny,Huard ^[2]

作者单位： Department of Orthopaedic Surgery, The Brown Foundation Institute of Molecular Medicine, McGovern Medical School, University of Texas Health Science Center at Houston, Houston, Texas. ^[1] Department of Orthopaedic Surgery, The Brown Foundation Institute of Molecular Medicine, McGovern Medical School, University of Texas Health Science Center at Houston, Houston, Texas.;Center for Regenerative Sports Medicine, Steadman Philippon Research Institute, Vail, Colorado. ^[2] Department of Biomedical Engineering, Rice University, Houston, Texas. ^[3] Orthopaedic Bioengineering Research Laboratory, Department of Mechanical Engineering and School of Biomedical Engineering, Colorado State University, Fort Collins, Colorado. ^[4]

关键词 MRL/MpJ mice osteoblasts osteoclasts ovariectomy tibia fracture