MSTN Attenuates Cardiac Hypertrophy through Inhibition of Excessive Cardiac Autophagy by Blocking AMPK /mTOR and miR-128/PPARγ/NF-κB.

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作者： Hanping,Qi ^[1] ; Jing,Ren ^[1] ; Lina,Ba ^[1] ; Chao,Song ^[1] ; Qianhui,Zhang ^[1] ; Yonggang,Cao ^[1] ; Pilong,Shi ^[1] ; Bowen,Fu ^[1] ; Yongsheng,Liu ^[1] ; Hongli,Sun ^[2]

作者单位： Department of Pharmacology, Harbin Medical University-Daqing, Daqing, Heilongjiang 163319, China. ^[1] Department of Pharmacology, Harbin Medical University-Daqing, Daqing, Heilongjiang 163319, China. Electronic address: hlsun2002@163.com. ^[2]

关键词 AMPK MSTN PPARγ autophagy cardiac hypertrophy miR-128

DOI 10.1016/j.omtn.2019.12.003

PMID 31923740

发布时间 2021-01-10

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Molecular therapy. Nucleic acids

2020年19卷

507-522页

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MSTN Attenuates Cardiac Hypertrophy through Inhibition of Excessive Cardiac Autophagy by Blocking AMPK /mTOR and miR-128/PPARγ/NF-κB.

Molecular therapy. Nucleic acids

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Molecular therapy. Nucleic acids

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MSTN Attenuates Cardiac Hypertrophy through Inhibition of Excessive Cardiac Autophagy by Blocking AMPK /mTOR and miR-128/PPARγ/NF-κB.

Molecular therapy. Nucleic acids

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Molecular therapy. Nucleic acids

相关期刊

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