Pretreatment with cilnidipine attenuates hypoxia/reoxygenation injury in HL-1 cardiomyocytes through enhanced NO production and action potential shortening.

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作者： Hiroyuki,Minato ^[1] ; Ichiro,Hisatome ^[2] ; Yasutaka,Kurata ^[3] ; Tomomi,Notsu ^[2] ; Naoe,Nakasone ^[4] ; Haruaki,Ninomiya ^[4] ; Toshihiro,Hamada ^[5] ; Takuya,Tomomori ^[6] ; Akihiro,Okamura ^[6] ; Junichiro,Miake ^[7] ; Motokazu,Tsuneto ^[2] ; Yasuaki,Shirayoshi ^[2] ; Ryo,Endo ^[1] ; Akihiro,Otsuki ^[1] ; Futoshi,Okada ^[8] ; Yoshimi,Inagaki ^[1]

作者单位： Department of Anesthesiology and Critical Care Medicine, Tottori University Faculty of Medicine, 86 Nishi-cho, Yonago, 683-8503, Japan. ^[1] Department of Genetic Medicine and Regenerative Therapeutics, Institute of Regenerative Medicine and Biofunction, Tottori University Graduate School of Medical Science, 86 Nishi-cho, Yonago, 683-8503, Japan. ^[2] Department of Physiology II, Kanazawa Medical University, Ishikawa, 920-0268, Japan. yasu@kanazawa-med.ac.jp. ^[3] Department of Biological Regulation, Tottori University, Yonago, 683-8503, Japan. ^[4] Department of Community-Based Family Medicine, Tottori University Faculty of Medicine, Yonago, 683-8503, Japan. ^[5] Department of Molecular Medicine and Therapeutics, Tottori University Faculty of Medicine, Yonago, 683-8503, Japan. ^[6] Department of Pharmacology, Tottori University Faculty of Medicine, Yonago, 683-8503, Japan. ^[7] Division of Pathological Biochemistry, Tottori University Faculty of Medicine, Yonago, 683-8503, Japan. ^[8]

关键词 Action potential Cilnidipine Ischemia reperfusion NO eNOS