Apolipoprotein E Deficiency Aggravates Neuronal Injury by Enhancing Neuroinflammation via the JNK/c-Jun Pathway in the Early Phase of Experimental Subarachnoid Hemorrhage in Mice.

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作者： Yue,Wu ^[1] ; Jinwei,Pang ^[2] ; Jianhua,Peng ^[2] ; Fang,Cao ^[3] ; Zongduo,Guo ^[1] ; Li,Jiang ^[1] ; Zhipeng,Teng ^[4] ; Zhijian,Huang ^[1] ; Chongjie,Cheng ^[1] ; Yong,Jiang ^[2] ; Xiaochuan,Sun ^[1]

作者单位： Department of Neurosurgery, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China. ^[1] Department of Neurosurgery, The Affiliated Hospital of Southwest Medical University, Luzhou, China. ^[2] Department of Cerebrovascular Disease, The Affiliated Hospital of Zunyi Medical College, Zunyi, China. ^[3] Department of Neurosurgery, Chongqing Traditional Chinese Medicine Hospital, Chongqing, China. ^[4]

主题词动物(Animals);载脂蛋白E类(Apolipoproteins E);细胞凋亡(Apoptosis);脑损伤(Brain Injuries);疾病模型, 动物(Disease Models, Animal);炎症(Inflammation);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);丝裂原活化蛋白激酶8(Mitogen-Activated Protein Kinase 8);神经元(Neurons);信号传导(Signal Transduction);蛛网膜下腔出血(Subarachnoid Hemorrhage)

DOI 10.1155/2019/3832648

PMID 31949876

发布时间 2020-06-29

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Oxidative medicine and cellular longevity

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Apolipoprotein E Deficiency Aggravates Neuronal Injury by Enhancing Neuroinflammation via the JNK/c-Jun Pathway in the Early Phase of Experimental Subarachnoid Hemorrhage in Mice.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

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Apolipoprotein E Deficiency Aggravates Neuronal Injury by Enhancing Neuroinflammation via the JNK/c-Jun Pathway in the Early Phase of Experimental Subarachnoid Hemorrhage in Mice.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

相关期刊

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