首页> Cell communication and signaling : CCS> Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components.

Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components.

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第一作者： Paula,Lindner

第一单位： Centre for Molecular Medicine Norway (NCMM), Nordic EMBL Partnership for Molecular Medicine, University of Oslo, P.O. Box 1137, Blindern, N-0318, Oslo, Norway.;Danish Research Institute of Translational Neuroscience (DANDRITE), Nordic EMBL Partnership for Molecular Medicine, Department of Molecular Biology and Genetics, Aarhus University, Aarhus, Denmark.

作者： Paula,Lindner ^[1] ; Søren Brøgger,Christensen ^[2] ; Poul,Nissen ^[3] ; Jesper Vuust,Møller ^[4] ; Nikolai,Engedal ^[5]

作者单位： Centre for Molecular Medicine Norway (NCMM), Nordic EMBL Partnership for Molecular Medicine, University of Oslo, P.O. Box 1137, Blindern, N-0318, Oslo, Norway.;Danish Research Institute of Translational Neuroscience (DANDRITE), Nordic EMBL Partnership for Molecular Medicine, Department of Molecular Biology and Genetics, Aarhus University, Aarhus, Denmark. ^[1] Department of Drug Design and Pharmacology, University of Copenhagen, Copenhagen, Denmark. ^[2] Danish Research Institute of Translational Neuroscience (DANDRITE), Nordic EMBL Partnership for Molecular Medicine, Department of Molecular Biology and Genetics, Aarhus University, Aarhus, Denmark. ^[3] Department of Biomedicine, Aarhus University, Aarhus, Denmark. ^[4] Centre for Molecular Medicine Norway (NCMM), Nordic EMBL Partnership for Molecular Medicine, University of Oslo, P.O. Box 1137, Blindern, N-0318, Oslo, Norway. k.n.engedal@ncmm.uio.no. ^[5]