Human BCL-G regulates secretion of inflammatory chemokines but is dispensable for induction of apoptosis by IFN-γ and TNF-α in intestinal epithelial cells.
作者:
主题词
细胞凋亡(Apoptosis);细胞系, 肿瘤(Cell Line, Tumor);趋化因子CCL20(Chemokine CCL20);趋化因子CCL5(Chemokine CCL5);炎症趋化因子类(Chemokines);结直肠肿瘤(Colorectal Neoplasms);上皮细胞(Epithelial Cells);基因敲除技术(Gene Knockout Techniques);人类(Humans);炎性肠疾病(Inflammatory Bowel Diseases);干扰素γ(Interferon-gamma);肠黏膜(Intestinal Mucosa);NF-κB(NF-kappa B);类器官(Organoids);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);STAT1转录因子(STAT1 Transcription Factor);转录因子(Transcription Factors);肿瘤坏死因子α(Tumor Necrosis Factor-alpha);增量调节(Up-Regulation)
DOI
10.1038/s41419-020-2263-0
PMID
31988296
发布时间
2021-01-26
- 浏览2
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文