The role of hepcidin and iron homeostasis in atherosclerosis.

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作者： Florian,Wunderer ^[1] ; Lisa,Traeger ^[2] ; Haakon H,Sigurslid ^[3] ; Patrick,Meybohm ^[4] ; Donald B,Bloch ^[5] ; Rajeev,Malhotra ^[6]

作者单位： Department of Anesthesiology, Intensive Care Medicine and Pain Therapy, University Hospital Frankfurt, Goethe University, Frankfurt, Germany. ^[1] Anesthesia Center for Critical Care Research of the Department of Anesthesia, Critical Care and Pain Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, United States. ^[2] Cardiovascular Research Center and the Cardiology Division of the Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, United States. ^[3] Department of Anesthesiology, Intensive Care Medicine and Pain Therapy, University Hospital Frankfurt, Goethe University, Frankfurt, Germany; Department of Anaesthesiology, University Hospital Wuerzburg, Wuerzburg, Germany. ^[4] Anesthesia Center for Critical Care Research of the Department of Anesthesia, Critical Care and Pain Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, United States; Division of Rheumatology, Allergy and Immunology of the Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, United States. ^[5] Cardiovascular Research Center and the Cardiology Division of the Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, United States. Electronic address: rmalhotra@mgh.harvard.edu. ^[6]

关键词 Atherosclerosis Bone morphogenetic proteins Hepcidin Inflammation Iron Macrophages