TP53 mutated AML subclones exhibit engraftment in a humanized bone marrow ossicle mouse model.

导出 OA

翻译打印收藏纠错

作者： Gabriel,Pabst ^[1] ; Karin,Lind ^[1] ; Ricarda,Graf ^[2] ; Armin,Zebisch ^[3] ; Friedrich,Stölzel ^[4] ; Konstanze,Döhner ^[5] ; Ellen,Heitzer ^[2] ; Andreas,Reinisch ^[6] ; Heinz,Sill ^[7]

作者单位： Division of Hematology, Department of Internal Medicine, Medical University of Graz, Auenbruggerplatz 38, 8036, Graz, Austria. ^[1] Institute of Human Genetics, Medical University of Graz, Graz, Austria. ^[2] Division of Hematology, Department of Internal Medicine, Medical University of Graz, Auenbruggerplatz 38, 8036, Graz, Austria.;Otto Loewi Research Center for Vascular Biology, Immunology and Inflammation, Division of Pharmacology, Medical University of Graz, Graz, Austria. ^[3] Department of Internal Medicine I, University Hospital Carl Gustav Carus, Dresden University of Technology Dresden, Dresden, Germany. ^[4] Department of Internal Medicine III, University Hospital of Ulm, Ulm, Germany. ^[5] Division of Hematology, Department of Internal Medicine, Medical University of Graz, Auenbruggerplatz 38, 8036, Graz, Austria. a.reinisch@medunigraz.at. ^[6] Division of Hematology, Department of Internal Medicine, Medical University of Graz, Auenbruggerplatz 38, 8036, Graz, Austria. heinz.sill@medunigraz.at. ^[7]