换一批Pro-inflammatory activation following demyelination is required for myelin clearance and oligodendrogenesis.
第一作者：
Maria Inês,Cunha
第一单位：
Institute of Neuronal Cell Biology, Technical University Munich, Munich, Germany.;German Center for Neurodegenerative Diseases, Munich, Germany.;Graduate Program in Areas of Basic and Applied Biology, Abel Salazar Biomedical Sciences Institute, University of Porto, Porto, Portugal.
作者：
医学主题词
动物(Animals);轴突(Axons);细胞, 培养的(Cells, Cultured);脱髓鞘疾病(Demyelinating Diseases);疾病模型, 动物(Disease Models, Animal);炎症(Inflammation);幼虫(Larva);溶血磷脂酰胆碱类(Lysophosphatidylcholines);小鼠(Mice);小神经胶质细胞(Microglia);突变(Mutation);髓鞘(Myelin Sheath);髓样分化因子88(Myeloid Differentiation Factor 88);少突神经胶质(Oligodendroglia);吞噬细胞(Phagocytes);吞噬体(Phagosomes);蛋白质组(Proteome);脊髓(Spinal Cord);肿瘤坏死因子α(Tumor Necrosis Factor-alpha);斑马鱼(Zebrafish)
DOI
10.1084/jem.20191390
PMID
32078678
发布时间
2020-12-24
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