换一批Ablation of glutaredoxin 1 promotes pulmonary angiogenesis and alveolar formation in hyperoxia-injured lungs by modifying HIF-1α stability and inhibiting the NF-κB pathway.
第一作者：
Xuwei,Liu
第一单位：
Department of Neonatology, Ministry of Education Key Laboratory of Child Development and Disorders, National Clinical Research Center for Child Health and Disorders, China International Science and Technology Cooperation Base of Child Development and Critical Disorders, Children's Hospital of Chongqing Medical University, Chongqing, PR China; Chongqing Key Laboratory of Pediatrics, PR China.
作者：
医学主题词
动物(Animals);基因缺失(Gene Deletion);谷氧还蛋白质类(Glutaredoxins);高氧症(Hyperoxia);缺氧诱导因子1, α亚基(Hypoxia-Inducible Factor 1, alpha Subunit);肺(Lung);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);NF-κB(NF-kappa B);新生血管化, 生理性(Neovascularization, Physiologic);蛋白质稳定性(Protein Stability);信号传导(Signal Transduction);血管内皮生长因子A(Vascular Endothelial Growth Factor A)
DOI
10.1016/j.bbrc.2020.02.129
PMID
32113683
发布时间
2020-11-11
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