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Actin-microtubule crosslinker Pod-1 tunes PAR-1 signaling to control synaptic development and tau-mediated synaptic toxicity.

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第一作者： Ha-Young,Kang

第一单位： Gwangju Center, Korea Basic Science Institute (KBSI), Gwangju, South Korea.

作者： Ha-Young,Kang ^[1] ; Hyung-Jun,Kim ^[2] ; Kiyoung,Kim ^[3] ; Seung-Il,Oh ^[1] ; Sunggyu,Yoon ^[4] ; Jaekwang,Kim ^[2] ; Sangwoo,Park ^[1] ; Yeongmi,Cheon ^[1] ; Song,Her ^[1] ; Mihye,Lee ^[5] ; Bingwei,Lu ^[6] ; Seongsoo,Lee ^[7]

作者单位： Gwangju Center, Korea Basic Science Institute (KBSI), Gwangju, South Korea. ^[1] Dementia Research Group, Korea Brain Research Institute (KBRI), Daegu, South Korea. ^[2] Department of Medical Biotechnology, Soonchunhyang University, Asan, South Korea. ^[3] Gwangju Center, Korea Basic Science Institute (KBSI), Gwangju, South Korea; Department of Life Science, College of Natural Science, Hanyang University, Seoul, South Korea. ^[4] Soonchunhyang Institute of Medi-bio Science, Soonchunhyang University, Cheonan, South Korea. Electronic address: mihyelee@sch.ac.kr. ^[5] Department of Pathology, Stanford University School of Medicine, Stanford, CA, USA. Electronic address: bingwei@stanford.edu. ^[6] Gwangju Center, Korea Basic Science Institute (KBSI), Gwangju, South Korea. Electronic address: soolee@kbsi.re.kr. ^[7]

关键词 Drosophila NMJ PAR-1 Pod-1 Synaptic defect Tau-mediated synaptic toxicity