首页> Proceedings of the National Academy of Sciences of the United States of America> Maternal activation of the EGFR prevents translocation of gut-residing pathogenic <i>Escherichia coli</i> in a model of late-onset neonatal sepsis.

Maternal activation of the EGFR prevents translocation of gut-residing pathogenic <i>Escherichia coli</i> in a model of late-onset neonatal sepsis.

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第一作者： Kathryn A,Knoop

第一单位： Department of Immunology, Mayo Clinic, Rochester, MN 55905; knoop.kathryn@mayo.edu.;Department of Internal Medicine, Washington University School of Medicine, St. Louis, MO 63110.

作者： Kathryn A,Knoop ^[1] ; Paige E,Coughlin ^[2] ; Alexandria N,Floyd ^[2] ; I Malick,Ndao ^[3] ; Carla,Hall-Moore ^[3] ; Nurmohammad,Shaikh ^[3] ; Andrew J,Gasparrini ^[4] ; Brigida,Rusconi ^[3] ; Marilyn,Escobedo ^[5] ; Misty,Good ^[3] ; Barbara B,Warner ^[3] ; Phillip I,Tarr ^[6] ; Rodney D,Newberry ^[2]

作者单位： Department of Immunology, Mayo Clinic, Rochester, MN 55905; knoop.kathryn@mayo.edu.;Department of Internal Medicine, Washington University School of Medicine, St. Louis, MO 63110. ^[1] Department of Internal Medicine, Washington University School of Medicine, St. Louis, MO 63110. ^[2] Department of Pediatrics, Washington University School of Medicine, St. Louis, MO 63110. ^[3] The Edison Family Center for Genome Sciences & Systems Biology, Washington University School of Medicine, St. Louis, MO 63110. ^[4] Department of Pediatrics, University of Oklahoma School of Medicine, Oklahoma City, OK 73019. ^[5] Department of Pediatrics, Washington University School of Medicine, St. Louis, MO 63110.;Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, MO 63110. ^[6]

关键词 bloodstream infections breast milk goblet cells neonate sepsis