换一批PMEPA1/TMEPAI isoforms function via its PY and Smad-interaction motifs for tumorigenic activities of breast cancer cells.
第一作者：
Meidi U,Puteri
第一单位：
Department of Experimental Pathology, Graduate School of Comprehensive Human Sciences and Faculty of Medicine, University of Tsukuba, Tsukuba, Japan.
作者：
医学主题词
氨基酸特征结构(Amino Acid Motifs);动物(Animals);乳腺肿瘤(Breast Neoplasms);COS细胞(COS Cells);细胞增殖(Cell Proliferation);女(雌)性(Female);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);基因敲除技术(Gene Knockout Techniques);HEK293细胞(HEK293 Cells);人类(Humans);膜蛋白质类(Membrane Proteins);类器官(Organoids);磷酸肌醇3-激酶(Phosphatidylinositol 3-Kinase);蛋白质亚型(Protein Isoforms);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction);Smad蛋白质类(Smad Proteins);转化生长因子β(Transforming Growth Factor beta)
DOI
10.1111/gtc.12766
PMID
32181976
发布时间
2020-10-19
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Genes to cells
375-390页
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