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首页> Aging> Overexpressed ITGA2 contributes to paclitaxel resistance by ovarian cancer cells through the activation of the AKT/FoxO1 pathway.

Overexpressed ITGA2 contributes to paclitaxel resistance by ovarian cancer cells through the activation of the AKT/FoxO1 pathway.

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作者： Linlin,Ma ^[1] ; Yan,Sun ^[2] ; Dan,Li ^[3] ; Hansong,Li ^[4] ; Xin,Jin ^[1] ; Dianyun,Ren ^[3]

作者单位： Department of Obstetrics and Gynecology, Beijing Hospital, National Center of Gerontology, Institute of Geriatric Medicine, Chinese Academy of Medical Sciences, Beijing 100730, R.P. China. ^[1] Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China. ^[2] Sino-German Laboratory of Personalized Medicine for Pancreatic Cancer, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China. ^[3] Cardiovascular Medicine Department, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China. ^[4] Cancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China. ^[5]

关键词 PTX resistance forkhead box O1 (FoxO1)integrin subunit α2 (ITGA2)p-AKT

主题词抗肿瘤药, 植物(Antineoplastic Agents, Phytogenic);细胞系, 肿瘤(Cell Line, Tumor);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);抗药性, 肿瘤(Drug Resistance, Neoplasm);女(雌)性(Female);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);整合素α2(Integrin alpha2);卵巢肿瘤(Ovarian Neoplasms);紫杉酚(Paclitaxel);磷酰化(Phosphorylation);预后(Prognosis);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction)

DOI 10.18632/aging.102954

PMID 32202508

发布时间 2021-12-13

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