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首页> Experimental neurology> Safinamide's potential in treating nondystrophic myotonias: Inhibition of skeletal muscle voltage-gated sodium channels and skeletal muscle hyperexcitability in vitro and in vivo.

Safinamide's potential in treating nondystrophic myotonias: Inhibition of skeletal muscle voltage-gated sodium channels and skeletal muscle hyperexcitability in vitro and in vivo.

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作者： Jean-François,Desaphy ^[1] ; Alessandro,Farinato ^[2] ; Concetta,Altamura ^[3] ; Michela,De Bellis ^[2] ; Paola,Imbrici ^[2] ; Nancy,Tarantino ^[2] ; Carla,Caccia ^[4] ; Elsa,Melloni ^[4] ; Gloria,Padoani ^[4] ; Silvia,Vailati ^[4] ; Charlotte,Keywood ^[4] ; Maria Rosaria,Carratù ^[3] ; Annamaria,De Luca ^[2] ; Diana,Conte ^[2] ; Sabata,Pierno ^[2]

作者单位： Department of Biomedical Sciences and Human Oncology, School of Medicine, University of Bari Aldo Moro, Bari, Italy. Electronic address: jeanfrancois.desaphy@uniba.it. ^[1] Department of Pharmacy & Drug Sciences, University of Bari Aldo Moro, Bari, Italy. ^[2] Department of Biomedical Sciences and Human Oncology, School of Medicine, University of Bari Aldo Moro, Bari, Italy. ^[3] Open R&D Department, Zambon S.p.A., Bresso, MI, Italy. ^[4]

关键词 Current-clamp In vivo model Muscle excitability Myotonia Patch-clamp Safinamide Voltage-gated sodium channels

主题词动作电位(Action Potentials);丙氨酸(Alanine);动物(Animals);苄胺类(Benzylamines);HEK293细胞(HEK293 Cells);人类(Humans);男(雄)性(Male);肌, 骨骼(Muscle, Skeletal);肌强直(Myotonia);大鼠(Rats);大鼠, Wistar(Rats, Wistar)

DOI 10.1016/j.expneurol.2020.113287

PMID 32205118

发布时间 2020-12-30

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