A naturally-occurring 22-bp coding deletion in Ugt86Dd reduces nicotine resistance in Drosophila melanogaster.

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作者： Stuart J,Macdonald ^[1] ; Chad A,Highfill ^[2]

作者单位： Department of Molecular Biosciences, University of Kansas, 4043 Haworth Hall, 1200 Sunnyside Avenue, Lawrence, KS, 66045, USA. sjmac@ku.edu. ^[1] Center for Computational Biology, University of Kansas, Lawrence, KS, 66047, USA. sjmac@ku.edu. ^[2] Department of Molecular Biosciences, University of Kansas, 4043 Haworth Hall, 1200 Sunnyside Avenue, Lawrence, KS, 66045, USA. ^[3]

关键词 CRISPR DSPR Drosophila Nicotine QTL Ugt35C1 Ugt86Dd

主题词动物(Animals);果蝇蛋白质类(Drosophila Proteins);黑腹果蝇(Drosophila melanogaster);抗药性(Drug Resistance);基因缺失(Gene Deletion);尼古丁(Nicotine);烟碱激动剂(Nicotinic Agonists)

DOI 10.1186/s13104-020-05035-z

PMID 32228671

发布时间 2022-07-20

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