Exercise improves cardiac function and glucose metabolism in mice with experimental myocardial infarction through inhibiting HDAC4 and upregulating GLUT1 expression.
第一作者:
Hao,Jiang
第一单位:
Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai, China.;Shanghai Institute of Cardiovascular Diseases, Shanghai, China.;NHC Key Laboratory of Viral Heart Diseases, Shanghai, China.;Key Laboratory of Viral Heart Diseases, Chinese Academy of Medical Sciences, Shanghai, China.
作者:
医学主题词
动物(Animals);疾病模型, 动物(Disease Models, Animal);能量代谢(Energy Metabolism);葡萄糖(Glucose);葡萄糖转运体1型(Glucose Transporter Type 1);组蛋白脱乙酰基酶类(Histone Deacetylases);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);心肌梗死(Myocardial Infarction);肌细胞, 心脏(Myocytes, Cardiac);身体条件作用, 动物(Physical Conditioning, Animal);增量调节(Up-Regulation)
DOI
10.1007/s00395-020-0787-1
PMID
32236769
发布时间
2021-06-25
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