Cellular Stress Upregulates Indole Signaling Metabolites in Escherichia coli.

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作者： Chung Sub,Kim ^[1] ; Jhe-Hao,Li ^[1] ; Brenden,Barco ^[2] ; Hyun Bong,Park ^[1] ; Alexandra,Gatsios ^[1] ; Ashiti,Damania ^[2] ; Rurun,Wang ^[3] ; Thomas P,Wyche ^[3] ; Grazia,Piizzi ^[3] ; Nicole K,Clay ^[2] ; Jason M,Crawford ^[4]

作者单位： Department of Chemistry, Yale University, New Haven, CT 06520, USA; Chemical Biology Institute, Yale University, West Haven, CT 06516, USA. ^[1] Department of Molecular, Cellular and Developmental Biology, Yale University, New Haven, CT 06511, USA. ^[2] Exploratory Science Center, Merck & Co., Inc., Cambridge, MA 02141, USA. ^[3] Department of Chemistry, Yale University, New Haven, CT 06520, USA; Chemical Biology Institute, Yale University, West Haven, CT 06516, USA; Department of Microbial Pathogenesis, Yale School of Medicine, New Haven, CT 06536, USA. Electronic address: jason.crawford@yale.edu. ^[4]

关键词 E.?coli IL-6 antibiotic tolerance host-bacteria interactions indole innate immunity persister cell signaling stress response

主题词动物(Animals);拟南芥属(Arabidopsis);大肠杆菌(Escherichia coli);粪便(Feces);人类(Humans);吲哚类(Indoles);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);分子结构(Molecular Structure);氧化性应激(Oxidative Stress);信号传导(Signal Transduction);增量调节(Up-Regulation)

DOI 10.1016/j.chembiol.2020.03.003

PMID 32243812

发布时间 2024-03-29

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Cell chemical biology

2020年27卷6期

698-707.e7页

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Cellular Stress Upregulates Indole Signaling Metabolites in Escherichia coli.

Cell chemical biology

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Cellular Stress Upregulates Indole Signaling Metabolites in Escherichia coli.

Cell chemical biology

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Cell chemical biology

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