SK4 calcium-activated potassium channels activated by sympathetic nerves enhances atrial fibrillation vulnerability in a canine model of acute stroke.

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作者： Mei,Yang ^[1] ; Youcheng,Wang ^[2] ; Xiaoxing,Xiong ^[3] ; Baojun,Xie ^[1] ; Jia,Liu ^[2] ; Junkui,Yin ^[3] ; Liuliu,Zi ^[4] ; Xi,Wang ^[5] ; Yanhong,Tang ^[4] ; Congxin,Huang ^[1] ; Qingyan,Zhao ^[2]

作者单位： Department of Cardiology, Renmin Hospital of Wuhan University, 238 Jiefang Road, Wuchang, Wuhan City, 430060, PR China. ^[1] Cardiovascular Research Institute of Wuhan University, 238 Jiefang Road, Wuchang, Wuhan City, 430060, PR China. ^[2] Hubei Key Laboratory of Cardiology, 238 Jiefang Road, Wuchang, Wuhan City, 430060, PR China. ^[3] Department of Neurosurgery, Renmin Hospital of Wuhan University, Wuhan City, 430060, PR China. ^[4] Department of Radiology, Renmin Hospital of Wuhan University, Wuhan City, 430060, PR China. ^[5]

关键词 Atrial fibrillation Cardiovascular system Circulatory system Intermediate-conductance KCa channels Internal medicine Laboratory medicine Neuroscience Pharmacology Stroke Sympathetic nerve

DOI 10.1016/j.heliyon.2020.e03928

PMID 32420493

发布时间 2022-04-14

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2020年6卷5期

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SK4 calcium-activated potassium channels activated by sympathetic nerves enhances atrial fibrillation vulnerability in a canine model of acute stroke.

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SK4 calcium-activated potassium channels activated by sympathetic nerves enhances atrial fibrillation vulnerability in a canine model of acute stroke.

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