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首页> Bioscience reports> ATP reduces mitochondrial MECR protein in liver of diet-induced obese mice in mechanism of insulin resistance.

ATP reduces mitochondrial MECR protein in liver of diet-induced obese mice in mechanism of insulin resistance.

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作者： Shengnan,Qian ^[1] ; Li,Ma ^[2] ; Shiqiao,Peng ^[2] ; Yanhong,Xu ^[3] ; Kaiyue,Wu ^[2] ; Shuang,Shen ^[4] ; Xiaoying,Zhang ^[2] ; Yongning,Sun ^[4] ; Jianping,Ye ^[1]

作者单位： College of Food Science and Technology, College of Fisheries and Life Science, Shanghai Ocean University, Shanghai 201306, China. ^[1] Central Laboratory, Shanghai Sixth People's Hospital East Campus, Shanghai University of Health and Medical Sciences, Shanghai 201306, China. ^[2] Department of Traditional Chinese Medicine, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai 200233, China. ^[3] Shanghai Diabetes Institute, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai 201306, China. ^[4] Department of Cardiology, Shanghai Municipal Hospital of Traditional Chinese Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 200071, China. ^[5]

关键词 ATP Berberine MECR hepatic physiology insulin resistance mitochondrial dysfunction

主题词 3T3-L1细胞(3T3-L1 Cells);腺苷三磷酸(Adenosine Triphosphate);动物(Animals);小檗碱(Berberine);疾病模型, 动物(Disease Models, Animal);减量调节(Down-Regulation);肝细胞(Hepatocytes);胰岛素(Insulin);胰岛素抗药性(Insulin Resistance);肝(Liver);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);线粒体, 肝(Mitochondria, Liver);肥胖症(Obesity);氧化还原酶类(作用于CH-CH键供体)(Oxidoreductases Acting on CH-CH Group Donors)

DOI 10.1042/BSR20200665

PMID 32440681

发布时间 2021-03-30

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2020年40卷6期

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