Wnt/β-catenin signaling contributes to prostate cancer heterogeneity through reciprocal suppression of H3K27 trimethylation.

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第一作者： Fan,Wang

第一单位： State Key Laboratory of Oncogenes and Related Genes, Renji-Med X Clinical Stem Cell Research Center, Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China.

作者： Fan,Wang ^[1] ; Yuli,Zhu ^[2] ; Fang,Wang ^[1] ; Yanqing,Wang ^[3] ; Bai-Jun,Dong ^[3] ; Naitao,Wang ^[4] ; Wei-Qiang,Gao ^[5]

作者单位： State Key Laboratory of Oncogenes and Related Genes, Renji-Med X Clinical Stem Cell Research Center, Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China. ^[1] Department of Ultrasound, Zhongshan Hospital, Fudan University, Shanghai, China; Shanghai Institute of Medical Imaging, Fudan University, Shanghai, China. ^[2] Department of Urology, Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China. ^[3] State Key Laboratory of Oncogenes and Related Genes, Renji-Med X Clinical Stem Cell Research Center, Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China. Electronic address: wangnaitao@sjtu.edu.cn. ^[4] State Key Laboratory of Oncogenes and Related Genes, Renji-Med X Clinical Stem Cell Research Center, Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; School of Biomedical Engineering & Med-X Research Institute, Shanghai Jiao Tong University, Shanghai, China. Electronic address: gao.weiqiang@sjtu.edu.cn. ^[5]

关键词 Cancer stem cell H3K27me3 Heterogeneity Prostate cancer Wnt/β-catenin signaling