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GSK-3β regulates the synaptic expression of NMDA receptors via phosphorylation of phosphatidylinositol 4 kinase type IIα.

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第一作者： Mascia,Amici

第一单位： Glutamate Receptor Group, School of Physiology, Pharmacology and Neuroscience, University of Bristol, Bristol, UK.

作者： Mascia,Amici ^[1] ; Yeseul,Lee ^[2] ; Robert J P,Pope ^[1] ; Clarrisa A,Bradley ^[3] ; Adam,Cole ^[4] ; Graham L,Collingridge ^[5]

作者单位： Glutamate Receptor Group, School of Physiology, Pharmacology and Neuroscience, University of Bristol, Bristol, UK. ^[1] Glutamate Receptor Group, School of Physiology, Pharmacology and Neuroscience, University of Bristol, Bristol, UK.;Lunenfeld-Tanenbaum Research Institute, Mount Sinai Hospital, Toronto, ON, Canada. ^[2] Neurosciences and Mental Health, Peter Gilgan Centre for Research and Learning, The Hospital for Sick Children, Toronto, ON, Canada. ^[3] Neurosignalling and Mood Disorders Group, Garvan Institute of Medical Research, Sydney, NSW, Australia. ^[4] Glutamate Receptor Group, School of Physiology, Pharmacology and Neuroscience, University of Bristol, Bristol, UK.;Lunenfeld-Tanenbaum Research Institute, Mount Sinai Hospital, Toronto, ON, Canada.;Tanz Centre for Research in Neurodegenerative Disease, Department of Physiology, The University of Toronto, Toronto, ON, Canada. ^[5]

关键词 GSK-3β NMDA receptors PI4KIIα long-term depression rat hippocampus

医学主题词 1-磷脂酰肌醇4-激酶(1-Phosphatidylinositol 4-Kinase);动物(Animals);糖原合成酶激酶3(Glycogen Synthase Kinase 3);海马(Hippocampus);磷酰化(Phosphorylation);大鼠(Rats);受体, N-甲基-D-天冬氨酸(Receptors, N-Methyl-D-Aspartate)

DOI 10.1111/ejn.14841

PMID 32463939

发布时间 2022-05-31

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