Silencing of CCR4-NOT complex subunits affects heart structure and function.

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作者： Lisa,Elmén ^[1] ; Claudia B,Volpato ^[2] ; Anaïs,Kervadec ^[1] ; Santiago,Pineda ^[1] ; Sreehari,Kalvakuri ^[1] ; Nakissa N,Alayari ^[1] ; Luisa,Foco ^[2] ; Peter P,Pramstaller ^[2] ; Karen,Ocorr ^[1] ; Alessandra,Rossini ^[2] ; Anthony,Cammarato ^[3] ; Alexandre R,Colas ^[1] ; Andrew A,Hicks ^[4] ; Rolf,Bodmer ^[5]

作者单位： Development Aging and Regeneration Program, Sanford Burnham Prebys Medical Discovery Institute, 10901 N Torrey Pines Rd, La Jolla, CA 92037, USA. ^[1] Institute for Biomedicine, Eurac Research, Affiliated Institute of the University of Lübeck, Via Galvani 31, 39100 Bolzano, Italy. ^[2] Johns Hopkins University, Division of Cardiology, 720 Rutland Ave., Baltimore, MD 21205, USA. ^[3] Institute for Biomedicine, Eurac Research, Affiliated Institute of the University of Lübeck, Via Galvani 31, 39100 Bolzano, Italy rolf@sbpdiscovery.org andrew.hicks@eurac.edu. ^[4] Development Aging and Regeneration Program, Sanford Burnham Prebys Medical Discovery Institute, 10901 N Torrey Pines Rd, La Jolla, CA 92037, USA rolf@sbpdiscovery.org andrew.hicks@eurac.edu. ^[5]

关键词 Arrhythmia CNOT1 Cardiomyocytes Drosophila heart GWAS Long-QT syndrome hiPSC