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首页> Cellular immunology> Gain-of-function mutations in CARD11 promote enhanced aggregation and idiosyncratic signalosome assembly.

Gain-of-function mutations in CARD11 promote enhanced aggregation and idiosyncratic signalosome assembly.

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作者： Jeffrey R,Stinson ^[1] ; Batsukh,Dorjbal ^[2] ; Dennis P,McDaniel ^[3] ; Liron,David ^[4] ; Hao,Wu ^[5] ; Andrew L,Snow ^[6]

作者单位： Department of Pharmacology & Molecular Therapeutics, Uniformed Services University of Health Sciences, Bethesda, MD, United States. Electronic address: jeffrey.stinson@nist.gov. ^[1] Department of Pharmacology & Molecular Therapeutics, Uniformed Services University of Health Sciences, Bethesda, MD, United States. ^[2] Biomedical Instrumentation Center, Uniformed Services University of Health Sciences, Bethesda, MD, United States. ^[3] Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, MA, United States. ^[4] Program in Cellular and Molecular Medicine, Boston Children's Hospital, Boston, MA, United States. ^[5] Department of Pharmacology & Molecular Therapeutics, Uniformed Services University of Health Sciences, Bethesda, MD, United States. Electronic address: andrew.snow@usuhs.edu. ^[6]

关键词 Aggregates B cell lymphoma BENTA CARD11 MALT1 NF-kB Signalosome

主题词凋亡调节蛋白质类(Apoptosis Regulatory Proteins);CARD信号接头蛋白质类(CARD Signaling Adaptor Proteins);细胞集合(Cell Aggregation);鸟苷酸环化酶(Guanylate Cyclase);人类(Humans);I-κB激酶(I-kappa B Kinase);Jurkat细胞(Jurkat Cells);淋巴细胞活化(Lymphocyte Activation);淋巴细胞(Lymphocytes);淋巴瘤, 大B细胞, 弥漫性(Lymphoma, Large B-Cell, Diffuse);NF-κB(NF-kappa B);信号传导(Signal Transduction)

DOI 10.1016/j.cellimm.2020.104129

PMID 32473470

发布时间 2024-07-31

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