Hypoxia-induced shift in the phenotype of proteasome from 26S toward immunoproteasome triggers loss of immunoprivilege of mesenchymal stem cells.

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第一作者： Ejlal,Abu-El-Rub

第一单位： Institute of Cardiovascular Sciences, St. Boniface General Hospital Research Centre, Regenerative Medicine Program, Department of Physiology and Pathophysiology, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, R2H2A6, Canada.

作者： Ejlal,Abu-El-Rub ^[1] ; Niketa,Sareen ^[1] ; Weiang,Yan ^[1] ; Keshav Narayan,Alagarsamy ^[1] ; Alireza,Rafieerad ^[1] ; Abhay,Srivastava ^[1] ; Vincenzo,Desiderio ^[2] ; Sanjiv,Dhingra ^[3]

作者单位： Institute of Cardiovascular Sciences, St. Boniface General Hospital Research Centre, Regenerative Medicine Program, Department of Physiology and Pathophysiology, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, R2H2A6, Canada. ^[1] Department of Experimental Medicine, University of Campania Luigi Vanvitelli, 80138, Naples, Italy. ^[2] Institute of Cardiovascular Sciences, St. Boniface General Hospital Research Centre, Regenerative Medicine Program, Department of Physiology and Pathophysiology, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, R2H2A6, Canada. sdhingra@sbrc.ca. ^[3]