Distinct iNKT Cell Populations Use IFNγ or ER Stress-Induced IL-10 to Control Adipose Tissue Homeostasis.

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第一作者： Nelson M,LaMarche

第一单位： Division of Rheumatology, Inflammation, and Immunity, Brigham and Women's Hospital, Boston, MA, USA; Program in Immunology, Harvard Medical School, Boston, MA, USA.

作者： Nelson M,LaMarche ^[1] ; Harry,Kane ^[2] ; Ayano C,Kohlgruber ^[3] ; Han,Dong ^[4] ; Lydia,Lynch ^[5] ; Michael B,Brenner ^[6]

作者单位： Division of Rheumatology, Inflammation, and Immunity, Brigham and Women's Hospital, Boston, MA, USA; Program in Immunology, Harvard Medical School, Boston, MA, USA. ^[1] Trinity Biomedical Science Institute, Trinity College Dublin, Dublin, Ireland. ^[2] Division of Genetics, Brigham and Women's Hospital, Boston, MA, USA. ^[3] Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, MA, USA; Department of Medicine, Brigham and Women's Hospital, Boston, MA, USA; Department of Microbiology and Immunology, Harvard Medical School, Boston, MA, USA. ^[4] Trinity Biomedical Science Institute, Trinity College Dublin, Dublin, Ireland; Division of Endocrinology, Diabetes, and Hypertension, Brigham and Women's Hospital, Boston, MA, USA. ^[5] Division of Rheumatology, Inflammation, and Immunity, Brigham and Women's Hospital, Boston, MA, USA; Program in Immunology, Harvard Medical School, Boston, MA, USA; Department of Medicine, Brigham and Women's Hospital, Boston, MA, USA. Electronic address: mbrenner@research.bwh.harvard.edu. ^[6]

关键词 ER stress NK cells adipose tissue iNKT cells inflammation lipids macrophages obesity