Redox States of Protein Cysteines in Pathways of Protein Turnover and Cytoskeleton Dynamics Are Changed with Aging and Reversed by Slc7a11 Restoration in Mouse Lung Fibroblasts.

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作者： Yuxuan,Zheng ^[1] ; Michael L,Merchant ^[1] ; Tom J,Burke ^[2] ; Jeffrey D,Ritzenthaler ^[3] ; Ming,Li ^[4] ; Adam E,Gaweda ^[2] ; Frederick W,Benz ^[2] ; Jesse,Roman ^[1] ; Walter H,Watson ^[4]

作者单位： Department of Pharmacology and Toxicology, University of Louisville School of Medicine, Louisville, KY 40202, USA. ^[1] Department of Medicine, Division of Nephrology and Hypertension, University of Louisville School of Medicine, Louisville, KY 40202, USA. ^[2] Department of Medicine, Division of Gastroenterology, Hepatology and Nutrition, University of Louisville School of Medicine, Louisville, 40202 KY, USA. ^[3] Department of Medicine, Division of Pulmonary, Allergy and Critical Care Medicine, Sidney Kimmel Medical College and Jane and Leonard Korman Respiratory Institute, Thomas Jefferson University, Philadelphia, 19107 PA, USA. ^[4]

主题词衰老(Aging);氨基酸转运系统y+(Amino Acid Transport System y+);动物(Animals);胱氨酸(Cystine);细胞骨架(Cytoskeleton);女(雌)性(Female);成纤维细胞(Fibroblasts);肺(Lung);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);氧化还原(Oxidation-Reduction)

DOI 10.1155/2020/2468986

PMID 32587657

发布时间 2022-05-10

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Oxidative medicine and cellular longevity

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Redox States of Protein Cysteines in Pathways of Protein Turnover and Cytoskeleton Dynamics Are Changed with Aging and Reversed by Slc7a11 Restoration in Mouse Lung Fibroblasts.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

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Redox States of Protein Cysteines in Pathways of Protein Turnover and Cytoskeleton Dynamics Are Changed with Aging and Reversed by Slc7a11 Restoration in Mouse Lung Fibroblasts.

Oxidative medicine and cellular longevity

相似文献

Oxidative medicine and cellular longevity

相关期刊

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