换一批
换一批In Mice Subjected to Chronic Stress, Exogenous cBIN1 Preserves Calcium-Handling Machinery and Cardiac Function.
作者：
关键词
AAV9, adeno-associated virus 9ANOVA, analysis of varianceAR, adrenergic receptorATPase, adenosine triphosphataseBW, body weightCAMKII, Ca2+/calmodulin-dependent protein kinaseCMV, cytomegalovirusDi-8-ANNEPs, 4-[2-[6-(Dioctylamino)-2-naphthalenyl]ethenyl]-1-(3-sulfopropyl)-pyridinium, inner saltEC, excitation contractionEDV, end diastolic volumeEF, ejection fractionGFP, green fluorescent proteinHF, heart failureHR, heart rateHT, heterozygoteHW, heart weightISO, isoproterenolLSD, least significant differenceLTCC, voltage-dependent L-type calcium channelLV, left ventricularLW, lung weightPBS, phosphate-buffered salinePKA, protein kinase APLN, phospholambanRWT, relative wall thicknessRyR, ryanodine receptorSD, standard deviationSEM, standard error of the meanSERCA2a, sarcoplasmic reticulum calcium ATPase pump 2aSR, sarcoplasmic reticulumSTORM, stochastic optical reconstruction microscopyTAC, transverse aortic constrictionTEM, transmission electron microscopyWT, wild typecBIN1, cardiac bridging integrator 1diastolic dysfunctionheart failurejSR, junctional sarcoplasmic reticulumpressure overloadsympathetic overdrivet-tubulet-tubule, transverse-tubulevg, vector genome
DOI
10.1016/j.jacbts.2020.03.006
PMID
32613144
发布时间
2020-09-28
- 浏览0
JACC. Basic to translational science
2020年5卷6期
561-578页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
