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CD74 is dispensable for development of chronic lymphocytic leukemia in <i>Eµ</i>-TCL1 transgenic mice.

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第一作者： Romy,Barthel

第一单位： University of Cologne, Department I of Internal Medicine, Center for Integrated Oncology Aachen Bonn Cologne Duesseldorf, Germany.;CECAD Center of Excellence on 'Cellular Stress Responses in Aging-Associated Diseases', Cologne, Germany.;CMMC Center of Molecular Medicine Cologne, Cologne, Germany.

作者： Romy,Barthel ^[1] ; Oleg,Fedorchenko ^[1] ; Tanja,Velmans ^[1] ; Natascha,Rosen ^[1] ; Phuong-Hien,Nguyen ^[1] ; Nina,Reinart ^[1] ; Alexandra,Florin ^[2] ; Marco,Herling ^[1] ; Michael,Hallek ^[1] ; Günter,Fingerle-Rowson ^[1]

作者单位： University of Cologne, Department I of Internal Medicine, Center for Integrated Oncology Aachen Bonn Cologne Duesseldorf, Germany.;CECAD Center of Excellence on 'Cellular Stress Responses in Aging-Associated Diseases', Cologne, Germany.;CMMC Center of Molecular Medicine Cologne, Cologne, Germany. ^[1] Institute of Pathology, University Hospital of Cologne, Cologne, Germany. ^[2]

关键词 B-cells CD74 CLL MIF TCL1

主题词动物(Animals);细胞凋亡(Apoptosis);细胞增殖(Cell Proliferation);白血病, 淋巴细胞, 慢性, B细胞(Leukemia, Lymphocytic, Chronic, B-Cell);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);原癌基因蛋白质类(Proto-Oncogene Proteins)

DOI 10.1080/10428194.2020.1791851

PMID 32667245

发布时间 2022-05-31

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