A cross-reactive human IgA monoclonal antibody blocks SARS-CoV-2 spike-ACE2 interaction.

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第一作者： Monir,Ejemel

第一单位： MassBiologics of the University of Massachusetts Medical School, Boston, MA, USA.

作者： Monir,Ejemel ^[1] ; Qi,Li ^[1] ; Shurong,Hou ^[2] ; Zachary A,Schiller ^[1] ; Julia A,Tree ^[3] ; Aaron,Wallace ^[1] ; Alla,Amcheslavsky ^[1] ; Nese,Kurt Yilmaz ^[2] ; Karen R,Buttigieg ^[3] ; Michael J,Elmore ^[3] ; Kerry,Godwin ^[3] ; Naomi,Coombes ^[3] ; Jacqueline R,Toomey ^[1] ; Ryan,Schneider ^[1] ; Anudeep S,Ramchetty ^[1] ; Brianna J,Close ^[4] ; Da-Yuan,Chen ^[4] ; Hasahn L,Conway ^[4] ; Mohsan,Saeed ^[4] ; Chandrashekar,Ganesa ^[1] ; Miles W,Carroll ^[3] ; Lisa A,Cavacini ^[5] ; Mark S,Klempner ^[6] ; Celia A,Schiffer ^[7] ; Yang,Wang ^[8]

作者单位： MassBiologics of the University of Massachusetts Medical School, Boston, MA, USA. ^[1] Biochemistry and Molecular Pharmacology, University of Massachusetts Medical School, Boston, MA, USA. ^[2] National Infection Service, Public Health England, Porton Down, Salisbury, Wiltshire, UK. ^[3] National Emerging Infectious Diseases Laboratories, Boston University, Boston, MA, USA. ^[4] MassBiologics of the University of Massachusetts Medical School, Boston, MA, USA. Lisa.Cavacini@umassmed.edu. ^[5] MassBiologics of the University of Massachusetts Medical School, Boston, MA, USA. Mark.Klempner@umassmed.edu. ^[6] Biochemistry and Molecular Pharmacology, University of Massachusetts Medical School, Boston, MA, USA. Celia.Schiffer@umassmed.edu. ^[7] MassBiologics of the University of Massachusetts Medical School, Boston, MA, USA. Yang.Wang@umassmed.edu. ^[8]