换一批Interleukin-22 deficiency alleviates doxorubicin-induced oxidative stress and cardiac injury via the p38 MAPK/macrophage/Fizz3 axis in mice.
第一作者：
Jing,Ye
第一单位：
Department of Cardiology, The People's Hospital of Guangxi Zhuang Autonomous Region, Nanning, 530021, China; Department of Cardiology, Renmin Hospital of Wuhan University, Cardiovascular Research Institute, Wuhan University, Hubei Key Laboratory of Cardiology, Wuhan, 430060, China.
作者：
医学主题词
动物(Animals);细胞凋亡(Apoptosis);多柔比星(Doxorubicin);人类(Humans);白细胞介素类(Interleukins);巨噬细胞(Macrophages);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);氧化性应激(Oxidative Stress);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1016/j.redox.2020.101636
PMID
32863209
发布时间
2024-09-30
- 浏览0
Redox biology
2020年36卷
101636页
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