Caveolin-1 stabilizes ATP7A, a copper transporter for extracellular SOD, in vascular tissue to maintain endothelial function.

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作者： Varadarajan,Sudhahar ^[1] ; Mustafa Nazir,Okur ^[2] ; John P,O'Bryan ^[3] ; Richard D,Minshall ^[4] ; David,Fulton ^[5] ; Masuko,Ushio-Fukai ^[6] ; Tohru,Fukai ^[7]

作者单位： Vascular Biology Center, Medical College of Georgia at Augusta University, Augusta, Georgia. ^[1] Department of Pharmacology and Toxicology, Medical College of Georgia at Augusta University, Augusta, Georgia. ^[2] Charlie Norwood Veterans Affairs Medical Center, Augusta, Georgia. ^[3] Laboratory of Molecular Gerontology, National Institute on Aging, National Institutes of Health, Baltimore, Maryland. ^[4] Department of Cell and Molecular Pharmacology and Experimental Therapeutics, Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina. ^[5] Ralph H. Johnson Veterans Affairs Medical Center, Charleston, South Carolina. ^[6] Departments of Anesthesiology and Pharmacology, University of Illinois at Chicago, Chicago, Illinois. ^[7] Department of Medicine (Cardiology), Medical College of Georgia at Augusta University, Augusta, Georgia. ^[8]

关键词 ATP7A SOD3 caveolin-1 endothelial function oxidative stress