换一批
换一批Inhaled H<sub>2</sub> or CO<sub>2</sub> Do Not Augment the Neuroprotective Effect of Therapeutic Hypothermia in a Severe Neonatal Hypoxic-Ischemic Encephalopathy Piglet Model.
作者：
主题词
酸中毒(Acidosis);投药, 吸入(Administration, Inhalation);动物(Animals);动物, 新生(Animals, Newborn);凋亡诱导因子(Apoptosis Inducing Factor);新生儿窒息(Asphyxia Neonatorum);脑损害, 慢性(Brain Damage, Chronic);脑源性神经营养因子(Brain-Derived Neurotrophic Factor);二氧化碳(Carbon Dioxide);半胱氨酸天冬氨酸蛋白酶3(Caspase 3);尾状核(Caudate Nucleus);大脑皮质(Cerebral Cortex);疾病模型, 动物(Disease Models, Animal);药物评价, 临床前(Drug Evaluation, Preclinical);脑电描记术(Electroencephalography);诱发电位, 视觉(Evoked Potentials, Visual);基因表达调控(Gene Expression Regulation);海马(Hippocampus);氢(Hydrogen);低温, 人工(Hypothermia, Induced);缺氧缺血, 脑(Hypoxia-Ischemia, Brain);神经组织蛋白质类(Nerve Tissue Proteins);神经保护药(Neuroprotective Agents);器官特异性(Organ Specificity);随机分配(Random Allocation);猪(Swine)
DOI
10.3390/ijms21186801
PMID
32948011
发布时间
2024-08-02
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