Autophagy mitigates ethanol-induced mitochondrial dysfunction and oxidative stress in esophageal keratinocytes.
第一单位:
Division of Gastroenterology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
作者:
主题词
动物(Animals);自噬(Autophagy);细胞系(Cell Line);细胞, 培养的(Cells, Cultured);食管(Esophagus);乙醇(Ethanol);女(雌)性(Female);角蛋白细胞(Keratinocytes);男(雄)性(Male);膜电位, 线粒体(Membrane Potential, Mitochondrial);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);微管相关蛋白质类(Microtubule-Associated Proteins);线粒体(Mitochondria);氧化性应激(Oxidative Stress);蛋白激酶类(Protein Kinases);阻遏蛋白质类(Repressor Proteins);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases)
DOI
10.1371/journal.pone.0239625
PMID
32966340
发布时间
2021-12-04
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PloS one
e0239625页
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