Dysregulated Phosphorylation of p53, Autophagy and Stemness Attributes the Mutant p53 Harboring Colon Cancer Cells Impaired Sensitivity to Oxaliplatin.

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作者： Lubna,Therachiyil ^[1] ; Javeria,Haroon ^[2] ; Fairooz,Sahir ^[1] ; Kodappully S,Siveen ^[1] ; Shahab,Uddin ^[1] ; Michal,Kulinski ^[1] ; Joerg,Buddenkotte ^[3] ; Martin,Steinhoff ^[1] ; Roopesh,Krishnankutty ^[1]

作者单位： Translational Research Institute, Academic Health System, Hamad Medical Corporation, Doha, Qatar. ^[1] Department of Pharmaceutical Sciences, College of Pharmacy, Qatar University, Doha, Qatar. ^[2] Department of Dermatology and Venereology, Hamad Medical Corporation, Doha, Qatar. ^[3] Department of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation-Education City, Doha, Qatar. ^[4] Department of Medicine, Weill Cornell Medicine, New York, NY, United States. ^[5] College of Medicine, Qatar University, Doha, Qatar. ^[6]

关键词 HCT 116 HT 29 autophagy colon cancer drug resistance oxaliplatin p53 therapy

DOI 10.3389/fonc.2020.01744

PMID 32984059

发布时间 2023-11-04

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Frontiers in oncology

2020年10卷

1744页

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Dysregulated Phosphorylation of p53, Autophagy and Stemness Attributes the Mutant p53 Harboring Colon Cancer Cells Impaired Sensitivity to Oxaliplatin.

Frontiers in oncology

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Frontiers in oncology

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Dysregulated Phosphorylation of p53, Autophagy and Stemness Attributes the Mutant p53 Harboring Colon Cancer Cells Impaired Sensitivity to Oxaliplatin.

Frontiers in oncology

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Frontiers in oncology

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