首页> Shock (Augusta, Ga.)> Hyper-Activation of Endogenous GLP-1 System to Gram-negative Sepsis Is Associated With Early Innate Immune Response and Modulated by Diabetes.

Hyper-Activation of Endogenous GLP-1 System to Gram-negative Sepsis Is Associated With Early Innate Immune Response and Modulated by Diabetes.

导出原文传递

翻译打印收藏纠错

第一作者： Olga,Bloch

第一单位： Diabetes and Autoimmunity Research Laboratory, Yitzhak Shamir Medical Center Affiliated to Sackler Medical School Tel Aviv University, Zerifin, Israel.

作者： Olga,Bloch ^[1] ; Sivan H,Perl ^[2] ; Tsilia,Lazarovitch ^[3] ; Dana,Zelnik-Yovel ^[2] ; Itamar,Love ^[2] ; Lior,Mendel-Cohen ^[2] ; Galina,Goltsman ^[2] ; Hadar,Flor ^[2] ; Micha J,Rapoport ^[4]

作者单位： Diabetes and Autoimmunity Research Laboratory, Yitzhak Shamir Medical Center Affiliated to Sackler Medical School Tel Aviv University, Zerifin, Israel. ^[1] Department 'C' of Internal Medicine, Yitzhak Shamir Medical Center Affiliated to Sackler Medical School Tel Aviv University, Zerifin, Israel. ^[2] Laboratory of Microbiology, Yitzhak Shamir Medical Center Affiliated to Sackler Medical School Tel Aviv University, Zerifin, Israel. ^[3] Diabetes and Autoimmunity Research Laboratory, Yitzhak Shamir Medical Center Affiliated to Sackler Medical School Tel Aviv University, Zerifin, Israel.;Department 'C' of Internal Medicine, Yitzhak Shamir Medical Center Affiliated to Sackler Medical School Tel Aviv University, Zerifin, Israel. ^[4]