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Imatinib improves insulin resistance and inhibits injury-induced neointimal hyperplasia in high fat diet-fed mice.

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第一作者： Prahalathan,Pichavaram

第一单位： Charlie Norwood VA Medical Center, Augusta, GA, USA; Center for Pharmacy and Experimental Therapeutics, University of Georgia College of Pharmacy, Augusta, GA, USA.

作者： Prahalathan,Pichavaram ^[1] ; Noha M,Shawky ^[2] ; Thomas J,Hartney ^[3] ; John Y,Jun ^[4] ; Lakshman,Segar ^[5]

作者单位： Charlie Norwood VA Medical Center, Augusta, GA, USA; Center for Pharmacy and Experimental Therapeutics, University of Georgia College of Pharmacy, Augusta, GA, USA. ^[1] Charlie Norwood VA Medical Center, Augusta, GA, USA; Center for Pharmacy and Experimental Therapeutics, University of Georgia College of Pharmacy, Augusta, GA, USA; Department of Pharmacology and Toxicology, Faculty of Pharmacy, Mansoura University, Egypt. ^[2] Charlie Norwood VA Medical Center, Augusta, GA, USA. ^[3] Division of Endocrinology, Diabetes, and Metabolism, Pennsylvania State University College of Medicine, Hershey, PA, USA; Department of Medicine, Pennsylvania State University College of Medicine, Hershey, PA, USA. ^[4] Charlie Norwood VA Medical Center, Augusta, GA, USA; Center for Pharmacy and Experimental Therapeutics, University of Georgia College of Pharmacy, Augusta, GA, USA; Department of Medicine, Pennsylvania State University College of Medicine, Hershey, PA, USA; Vascular Biology Center, Department of Pharmacology and Toxicology, Augusta University, Augusta, GA, USA. Electronic address: lsegar@augusta.edu. ^[5]

关键词 Adiponectin Arterial injury Imatinib Insulin resistance Neointimal hyperplasia PDGF