换一批
换一批Prenatal indole-3-carbinol administration activates aryl hydrocarbon receptor-responsive genes and attenuates lung injury in a bronchopulmonary dysplasia model.
作者：
主题词
动物(Animals);动物, 新生(Animals, Newborn);支气管肺发育不良(Bronchopulmonary Dysplasia);细胞因子类(Cytokines);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);纤维化(Fibrosis);高氧症(Hyperoxia);吲哚类(Indoles);炎症介导素类(Inflammation Mediators);肺(Lung);肺损伤(Lung Injury);妊娠(Pregnancy);产前暴露迟发效应(Prenatal Exposure Delayed Effects);RNA, 信使(RNA, Messenger);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, 芳基烃(Receptors, Aryl Hydrocarbon);体重增长(Weight Gain);大鼠(Rats)
DOI
10.1177/1535370220963789
PMID
33148012
发布时间
2024-02-26
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