Deletion of mitochondrial uncoupling protein 2 exacerbates mitophagy and cell apoptosis after cerebral ischemia and reperfusion injury in mice.
第一作者:
Maotao,He
第一单位:
Department of Pathology, General Hospital of Ningxia Medical University, Yinchuan, Ningxia 750004, China.;School of Basic Medical Sciences, Department of Pathology, Ningxia Medical University; Ningxia Key Laboratory of Vascular Injury and Repair, Yinchuan, Ningxia 750004, China.;Department of Pharmaceutical Sciences, Biomanufacturing Research Institute and Technological Enterprise (BRITE), College of Health and Sciences, North Carolina Central University, Durham, NC 27707, USA.
作者:
主题词
动物(Animals);细胞凋亡(Apoptosis);脑(Brain);脑缺血(Brain Ischemia);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);人类(Humans);梗死, 大脑中动脉(Infarction, Middle Cerebral Artery);男(雄)性(Male);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);线粒体(Mitochondria);氧化性应激(Oxidative Stress);活性氧(Reactive Oxygen Species);再灌注损伤(Reperfusion Injury)
DOI
10.7150/ijms.49849
PMID
33162815
发布时间
2021-08-02
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