Abnormal podocyte TRPML1 channel activity and exosome release in mice with podocyte-specific Asah1 gene deletion.
第一作者:
Guangbi,Li
第一单位:
Department of Pharmacology and Toxicology, School of Medicine, Virginia Commonwealth University, Richmond, VA, USA.
作者:
主题词
酸性神经酰胺酶(Acid Ceramidase);动物(Animals);细胞, 培养的(Cells, Cultured);疾病模型, 动物(Disease Models, Animal);外泌体(Exosomes);人类(Humans);溶酶体(Lysosomes);男(雄)性(Male);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);肾病综合征(Nephrotic Syndrome);足细胞(Podocytes);瞬时受体电位通道(Transient Receptor Potential Channels);尿(Urine)
DOI
10.1016/j.bbalip.2020.158856
PMID
33221496
发布时间
2022-02-02
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Biochimica et biophysica acta. Molecular and cell biology of lipids
2021年1866卷2期
158856页
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