NCS 613, a PDE4 inhibitor, by increasing cAMP level suppresses systemic inflammation and immune complexes deposition in kidney of MRL/lpr lupus- prone mice.

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作者： Issaka,Yougbare ^[1] ; Thérèse,Keravis ^[2] ; Claire,Lugnier ^[3]

作者单位： UMR CNRS 7213, Biophotonics and Pharmacology, Faculty of Pharmacy, University of Strasbourg, 67401 Illkirch Cedex, France. Electronic address: issaka.yougbare@sanofi.com. ^[1] UMR CNRS 7213, Biophotonics and Pharmacology, Faculty of Pharmacy, University of Strasbourg, 67401 Illkirch Cedex, France. ^[2] UMR CNRS 7213, Biophotonics and Pharmacology, Faculty of Pharmacy, University of Strasbourg, 67401 Illkirch Cedex, France; Institute of Physiology, FMTS-EA 3072, Faculty of Medicine, University of Strasbourg, 11 Humann Street, 67000 Strasbourg, France. ^[3]

关键词 Autoantibodies Glomeruli nephritis NCS 613 PDE4 cAMP

主题词腺嘌呤(Adenine);动物(Animals);消炎药(Anti-Inflammatory Agents);抗原抗体复合物(Antigen-Antibody Complex);环AMP(Cyclic AMP);女(雌)性(Female);炎症(Inflammation);肾(Kidney);红斑狼疮, 系统性(Lupus Erythematosus, Systemic);狼疮肾炎(Lupus Nephritis);小鼠, 近交MRL lpr(Mice, Inbred MRL lpr);磷酸二酯酶4抑制剂(Phosphodiesterase 4 Inhibitors);小鼠(Mice)

DOI 10.1016/j.bbadis.2020.166019

PMID 33227426

发布时间 2024-02-26

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Biochimica et biophysica acta. Molecular basis of disease

2021年1867卷3期

166019页

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NCS 613, a PDE4 inhibitor, by increasing cAMP level suppresses systemic inflammation and immune complexes deposition in kidney of MRL/lpr lupus- prone mice.

Biochimica et biophysica acta. Molecular basis of disease

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Biochimica et biophysica acta. Molecular basis of disease

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NCS 613, a PDE4 inhibitor, by increasing cAMP level suppresses systemic inflammation and immune complexes deposition in kidney of MRL/lpr lupus- prone mice.

Biochimica et biophysica acta. Molecular basis of disease

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Biochimica et biophysica acta. Molecular basis of disease

相关期刊

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