Long non-coding maternally expressed gene 3 regulates cigarette smoke extract-induced apoptosis, inflammation and cytotoxicity by sponging miR-181a-2-3p in 16HBE cells.

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作者： Shiming,Fan ^[1] ; Yan,Ren ^[2] ; Wenli,Zhang ^[3] ; Huawei,Zhang ^[4] ; Cheng,Wang ^[5]

作者单位： Department of Respiratory Medicine, Changning County Hospital of Traditional Chinese Medicine, Yibin, Sichuan 644300, P.R. China. ^[1] Department of Pediatrics, Changning County Hospital of Traditional Chinese Medicine, Yibin, Sichuan 644300, P.R. China. ^[2] Department of Gastroenterology, Changning County Hospital of Traditional Chinese Medicine, Yibin, Sichuan 644300, P.R. China. ^[3] Department of Critical Care Medicine, Changning County Hospital of Traditional Chinese Medicine, Yibin, Sichuan 644300, P.R. China. ^[4] Department of Nephrology, Changning County Hospital of Traditional Chinese Medicine, Yibin, Sichuan 644300, P.R. China. ^[5]

关键词 apoptosis cell viability chronic obstructive pulmonary disease cytotoxicity inflammation maternally expressed gene 3 microR-181a-2-3p

DOI 10.3892/ol.2020.12306

PMID 33262837

发布时间 2020-12-07

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